MOTS-C
Endurance, metabolism & performance.
30-day full guarantee. Notice a difference or it's free — no return required.
What it does
Mechanism
Mitochondrial signal
MOTS-c is encoded in mitochondrial DNA and released from muscle during metabolic stress and exercise — an endogenous regulator of metabolism.
AMPK activation
It switches on AMPK, improving glucose uptake, fat oxidation and mitochondrial efficiency — the same signals exercise triggers.
Metabolic flexibility
The result is better fuel use at rest and under exertion; circulating MOTS-c declines with age and metabolic dysfunction.
The Evidence
These findings describe peer-reviewed research on MOTS-C — the actives in Peak Pulse. Claims are about the compounds, not our formulation.
Lee et al. · Cell Metabolism 2015
The founding study: MOTS-c promotes metabolic homeostasis and reduces insulin resistance via AMPK activation (diet-induced obese mice).
PMID 25738459.
Zheng & Wang · Frontiers in Endocrinology 2023
Review synthesising MOTS-c evidence across insulin resistance, ageing and cardiovascular health.
PMID 36761202.
Evidence is predominantly preclinical (rodent/cell); human data is observational so far. Studied in preclinical models, not proven in human trials. WADA-prohibited in sport.
The Product
Your Peak Pulse pen, a 30-day supply of single-use needle tips, and bacteriostatic water for reconstitution — one discreet, unbranded box.
Questions
Ships as lyophilised powder in a sterile pen. Reconstitute once with the bacteriostatic water supplied, then a quick subcutaneous injection. Ships stable at ambient temperature; refrigerate on arrival.
Supplied for research and educational purposes. Not TGA-approved and not sold as a medicine. Australian adults 18+ only.
Standard workplace and roadside screens test for recreational substances — peptides don't appear on them. If you compete under WADA rules, check the current prohibited list before use.
30-day full refund if you don't notice a difference. No return required.
